A recent study in PLOS Biology reveals that a low-dose combination of Zinc, BCAAs, and Serine synergistically reverses behavioral deficits in multiple autism mouse models. By restoring synaptic protein levels and normalizing "noisy" neural firing in the amygdala, the treatment effectively improved social behavior and memory. This suggests a scalable metabolic strategy to treat diverse neurodevelopmental disorders by targeting shared circuit dysfunctions rather than specific genes.